AP-1 proteins modulate unwanted fat accumulation in liver.

The CNIO study team proposes that the imbalance between the different AP-1 proteins represents a key part of FLD pathogenesis.. AP-1 proteins modulate unwanted fat accumulation in liver, say researchers Excessive alcohol consumption, in addition to obesity leads to the accumulation of excess fat in the liver, an illness termed fatty liver disease or steatosis. FLD is among the most prevalent diseases in Western societies and impacts about 30 percent of the adult population.Related StoriesProtein sensor for proprioception foundInner ear canal damage brain warnings from nerve cellsImproved outcomes yielded from new ChIP-seq protocolThe TSCs were also found to market survival of another cell type when transplanted jointly, suggesting that these cells may be able to modify their regional environment and enhance the survival of co-transplanted cells. Thus a fresh stem cell structured trophoblastic approach to therapeutic cell transplantation may prove to be beneficial.