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Alexandre Loupy, M.D buy generic viagra ., Ph.D., Carmen Lefaucheur, M.D., Ph.D., Dewi Vernerey, M.P.H., Christof Prugger, M.D., Jean-Paul Duong van Huyen, M.D., Ph.D., Nuala Mooney, Ph.D., Caroline Suberbielle, M.D., Ph.D.D., Ph.D.D.D., Dany Anglicheau, M.D., Ph.D., Dominique Nochy, M.D., Dominique Charron, M.D., Ph.D., Jean-Philippe Empana, M.D., Ph.D., Michel Delahousse, M.D., Christophe Legendre, M.D., Denis Glotz, M.D., Ph.D., Gary S. Hill, M.D., Adriana Zeevi, Ph.D., and Xavier Jouven, M.D., Ph.D.: Complement-Binding Anti-HLA Kidney-Allograft and Antibodies Survival Despite significant advances in transplantation, the induced alloimmune response continues to be a major determinant lately kidney-allograft loss.1-3 In the United Europe and States, thousands of kidney transplants fail every year, and kidney-allograft failure is a major reason behind end-stage renal disease, leading to increased morbidity, mortality, and costs.4,5 One of the most important advances in transplantation medicine has been the recognition that anti-HLA antibodies are destructive.6-10 Numerous studies over the past decade have indicated that the alloimmune response, mediated by anti-HLA antibodies, plays a key function in the failure of kidney allografts; this concept has been prolonged to center, lung, and composite tissue transplants.6 Although anti-HLA antibodies are considered to be harmful, there exists a wide spectrum of graft injury related to these antibodies, ranging from no recognizable damage to florid rejection.11,12 Such a varied effect underscores the need to define distinct graft phenotypes and outcomes based on the presence or absence and features of donor-specific anti-HLA antibodies after transplantation.

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